iNOS contributes to heart failure with preserved ejection fraction through mitochondrial dysfunction and Akt S-nitrosylation_北京益仁恒业科技有限公司

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iNOS contributes to heart failure with preserved ejection fraction through mitochondrial dysfunction and Akt S-nitrosylation

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1970-01-01

Guo Y, Wen J, He A, Qu C, Peng Y, Luo S, Wang X. iNOS contributes to heart failure with preserved ejection fraction through mitochondrial dysfunction and Akt S-nitrosylation. J Adv Res. 2023 Jan;43:175-186. doi: 10.1016/j.jare.2022.03.003. Epub 2022 Mar 5.

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Donor MSCs release apoptotic bodies to improve myocardial infarction via autophagy regulation in recipient cells

Fatty acid-binding protein 5 aggravates pulmonary artery fibrosis in pulmonary hypertension secondary to left heart disease via activating wnt/β-catenin pathway

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上一个：

Donor MSCs release apoptotic bodies to improve myocardial infarction via autophagy regulation in recipient cells

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Fatty acid-binding protein 5 aggravates pulmonary artery fibrosis in pulmonary hypertension secondary to left heart disease via activating wnt/β-catenin pathway

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α-myosin heavy chain lactylation maintains sarcomeric structure and function and alleviates the development of heart failure

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